Septic shock is associated with excessive sympathetic stimulation that can result in cardiac dysfunction and tachycardia. Despite beta-blockers historically being considered a relative contraindication in sepsis, recent literature has suggested that ultrashort acting beta-blockers may be an effective treatment option in controlling sepsis related tachycardia as well as overcoming the negative effects of excessive sympathetic stimulation. Please join Dr. Smith for a virtual presentation as he discusses the specific mechanisms in which sepsis and excessive sympathetic stimulation can result in cardiac dysfunction and the potential role ultrashort beta-blockers can have to overcome these negative effects. This session will focus on analyzing recent literature examining beta-blocker therapy in sepsis to identify patient characteristics that may see benefits or harms in receiving beta-blocker therapy.
To learn more about this activity, including speaker information and learning objectives, expand the module below (click the +). Pharmacists can earn 0.75 contact hour of knowledge-based CE credit at the completion of this activity.
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This 45 minute presentation will examine the specific mechanisms in which sepsis and excessive sympathetic stimulation can result in cardiac dysfunction and discuss the potential role ultrashort beta-blockers can have to overcome these negative effects. In addition, the session will focus on analyzing recent literature examining beta-blocker therapy in sepsis to identify patient characteristics that may see benefits or harms in receiving beta-blocker therapy.
- Describe the mechanisms in which sepsis can induce cardiac dysfunction
- Summarize recent literature evaluating beta-blocker therapy in patients with sepsis
- Identify patient specific characteristics that may see the most benefit or harm from starting beta-blocker therapy in sepsis
Jesse Smith, Pharm.D.
Release Date: Jan 11, 2022
Credit Expiration Date: Jan 11, 2025